NSAIDs and Acute Kidney Injury
Drug Induced Kidney Disease: Symptoms, Risk, Diagnosis, Treatment
Kidney damage from nonsteroidal anti‐inflammatory drugs—Myth or truth? Review of selected literature - Drożdżal - 2021 - Pharmacology Research & Perspectives - Wiley Online Library
Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
PDF) Diclofenac induced acute renal failure in a decompensated elderly patient
Biomedicines | Free Full-Text | A Single Oral Dose of Diclofenac Causes Transition of Experimental Subclinical Acute Kidney Injury to Chronic Kidney Disease
PDF) Diclofenac-Induced Kidney Damage in Wistar Rats: Involvement of Antioxidant Mechanism
Biomedicines | Free Full-Text | A Single Oral Dose of Diclofenac Causes Transition of Experimental Subclinical Acute Kidney Injury to Chronic Kidney Disease
NSAIDs in CKD: Are They Safe? - American Journal of Kidney Diseases
Topical Nonsteroidal Anti-inflammatory Drugs and Nephrotoxicity: Is There a Safer Option?
NSAIDs and blood pressure medicines | Healthify
Mitigative effect of caffeine against diclofenac-induced hepato-renal damage and chromosomal aberrations in male albino rats | BMC Complementary Medicine and Therapies | Full Text
Of NSAIDs, ibuprofen poses lowest risk for kidney function decline
Biomedicines | Free Full-Text | A Single Oral Dose of Diclofenac Causes Transition of Experimental Subclinical Acute Kidney Injury to Chronic Kidney Disease
Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
Protective effect of curcumin on the kidney of diclofenac sodium-challenged mice: apoptotic, redox potential and histopathological outcomes | The Journal of Basic and Applied Zoology | Full Text
Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
![PDF] Diclofenac-Induced Kidney Damage in Wistar Rats: Involvement of Antioxidant Mechanism | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/1854a56aa7355e1d4987dadd5c61e5d33763876f/7-Figure1-1.png "PDF] Diclofenac-Induced Kidney Damage in Wistar Rats: Involvement of Antioxidant Mechanism | Semantic Scholar")
PDF] Diclofenac-Induced Kidney Damage in Wistar Rats: Involvement of Antioxidant Mechanism | Semantic Scholar
Diclofenac-Induced Kidney Damage in Wistar Rats: Involvement of Antioxidant Mechanism
Protective effect of curcumin on the kidney of diclofenac sodium-challenged mice: apoptotic, redox potential and histopathological outcomes | The Journal of Basic and Applied Zoology | Full Text
Cancers | Free Full-Text | Development and Challenges of Diclofenac-Based Novel Therapeutics: Targeting Cancer and Complex Diseases
Pentoxifylline and berberine mitigate diclofenac-induced acute nephrotoxicity in male rats via modulation of inflammation and oxidative stress - ScienceDirect
Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
Biomedicines | Free Full-Text | A Single Oral Dose of Diclofenac Causes Transition of Experimental Subclinical Acute Kidney Injury to Chronic Kidney Disease
